1314687-82-5 Usage
General Description
6-Bromoquinoline-3-carbonitrile is an organic compound with the molecular formula C10H5BrN2. It is a derivative of quinoline and contains a bromine atom and a nitrile group. This chemical is commonly used as a building block or intermediate in organic synthesis, particularly in the pharmaceutical industry for the production of various drugs and pharmaceutical compounds. It has also been studied for its potential biological and medicinal properties, including as an anti-tumor agent. 6-Bromoquinoline-3-carbonitrile is a versatile compound with potential applications in various fields, making it a valuable chemical for research and industrial purposes.
Check Digit Verification of cas no
The CAS Registry Mumber 1314687-82-5 includes 10 digits separated into 3 groups by hyphens. The first part of the number,starting from the left, has 7 digits, 1,3,1,4,6,8 and 7 respectively; the second part has 2 digits, 8 and 2 respectively.
Calculate Digit Verification of CAS Registry Number 1314687-82:
(9*1)+(8*3)+(7*1)+(6*4)+(5*6)+(4*8)+(3*7)+(2*8)+(1*2)=165
165 % 10 = 5
So 1314687-82-5 is a valid CAS Registry Number.
1314687-82-5Relevant articles and documents
Ru(III)-catalyzed construction of variously substituted quinolines from 2-aminoaromatic aldehydes (ketones) and isoxazoles: Isoxazoles as cyclization reagent and cyano sources
Cui, Xiuling,Han, Xiliang,Hu, Di,Hu, Wei,Pi, Chao,Wu, Yangjie
supporting information, (2022/01/28)
A Ru(Ⅲ)-catalyzed annulation reaction of 2-aminoaromatic aldehydes (ketones) and isoxazoles to afford diverse 3-cyanoquinolines has been developed. Notably, isoxazole acted as a cyclization reagent and nontoxic cyano source via N-O bond cleavage and fragm
COMBINATION OF KINASE INHIBITORS AND USES THEREOF
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, (2015/02/19)
The present invention provides for a method for treating a disease condition associated with PI3-kinase a and/or a receptor tyrosine kinase (RTK) in a subject. In another aspect, the invention provides for a method for treating a disease condition associated with PI3-kinase α and/or an RTK in a subject. In yet another aspect, a method of inhibiting phosphorylation of Akt (S473) in a cell is set forth.
